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The Temporomandibular Joint
The temporomandibular joint is
unique. It is a
ginglymo-diarthrodial joint, which
is also linked to its contra-lateral
counterpart. Such daunting philology
suggests complexity and this indeed
is the case.
In general, there are two groups of
patients with temporomandibular
joint disorders. Those with normal
anatomy, but abnormal function, and
those with abnormal anatomy whose
function may be abnormal.
Temporomandibular joint dysfunction
is ill-understood, but may affect as
many as 40% of the population at
some time and is more common in
females. It may begin in adolescence
with pain and clicking in the joints
which often recovers, never to
recur. A small group have further
problems, some continuing into early
adult life before symptoms subside.
A very small percentage of these
develop increasing symptoms in their
40's and 50's and may end with
chronic facial pain. A second group
does not recover after the first
episode and eventually develops
continuous discomfort which may
profoundly upset their lives. It is
this group for whom various
hypotheses have been formulated.
Some regard the dental occlusion as
the "third joint surface" and
postulate that abnormalities in the
way teeth fit together generate
disharmony in movement of the joints
with symptoms caused by muscle
spasm, made worse by emotional upset
which can produce an increase in
muscle tone.
Psychological causes and stress can
increase temporomandibular joint
symptoms and TMJ dysfunction
patients have higher catecholamine
levels than controls and indeed
treatment with anti-depressants or
sedation improves many of these
patients.
Many feel this may be the basis of
dysfunction symptoms with pain
thought to be produced by
masticatory muscle spasm. Abnormal
habits, playing wind instruments,
occlusal disharmony,
over-contraction and fatigue of
muscles influence it. Conservative
management of the condition includes
exercises, advice about diet,
altering the dental bite with
splints and sedation or
anti-depressants.
Whichever the theory followed,
treatment involves conservative
measures first and about 40-50% of
patients will be improved by these
alone.
The second main group of patients
are those with abnormal anatomy. The
simplest is meniscus displacement
and in such patients plain
radiography is often of little use
if there is no hard tissue
abnormality. CT scanning is similar,
if more accurate. MRI scanning will
demonstrate the position of the
meniscus and shows bony tissues of
the joint.
Surgical treatment on these patients
must only be undertaken after very
careful evaluation and trial of
conservative treatment. Surgery can
be divided into two types:
reparative and reconstructive.
Repair involves restoring the
meniscus to its correct position,
repairing it if necessary. In the
past, surgery for TMJ disease was
less scientific and the results were
appropriately variable, but now
there is deeper understanding,
better investigation and
sub-specialisation of surgeons which
appears to improve outcome.
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